In our article recently published in J Steroid Biochem Mol Biol – a collaboration with Tytgat Institute for Liver and Intestinal Research (Amsterdam UMC, University of Amsterdam), Hospital La Fe and Hospital Manises – we show that treatment with vitamin D increases levels of its receptor and attenuates the proliferation of fibroblasts in Crohn’s disease. The Vitamin D Receptor Taq I Polymorphism Is Associated With Reduced VDR and Increased PDIA3 Protein Levels in Human Intestinal Fibroblasts Fibroblast viewed through a miscroscope
Succinate Activates EMT in Intestinal Epithelial Cells through SUCNR1: A Novel Protagonist in Fistula Development.
Diminished Vitamin D Receptor Protein Levels in Crohn’s Disease Fibroblasts: Effects of VD.